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dc.contributor.authorUstek, Duran
dc.contributor.authorEkmekci, Ahmet
dc.contributor.authorEren, Mehmet
dc.contributor.authorAbaci, Neslihan
dc.contributor.authorGulec, Cagri
dc.contributor.authorOzcan, Kazim Serhan
dc.contributor.authorGungor, Baris
dc.contributor.authorOsmonov, Damirbek
dc.contributor.authorZencirci, Aycan
dc.contributor.authorToprak, Ercan
dc.contributor.authorDonmez, Cevdet
dc.date.accessioned2021-03-04T18:09:55Z
dc.date.available2021-03-04T18:09:55Z
dc.date.issued2013
dc.identifier.citationEkmekci A., Ozcan K. S. , Gungor B., Abaci N., Osmonov D., Zencirci A., Toprak E., Donmez C., Ustek D., Gulec C., et al., "The relationship between endothelial nitric oxide synthase 4a/4b gene polymorphism and premature coronary artery disease", ACTA CARDIOLOGICA, cilt.68, ss.464-468, 2013
dc.identifier.issn0001-5385
dc.identifier.otherav_89463540-90fc-4dc8-a8ec-d6807e1e8c2b
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/93100
dc.identifier.urihttps://doi.org/10.1080/ac.68.5.2994468
dc.description.abstractBackground Nitric oxide (NO) plays a major role in the regulation of endothelial functions and reduced NO synthesis has been implicated in the development of coronary atherosclerosis. Endothelial nitric oxide synthase (eNOS) intron 4a/b polymorphism has been shown to be related to plasma nitric oxide concentrations and coronary artery disease in various population studies. The aim of this study is to assess the relationship between eNOS 4a/b polymorphism and premature CAD.
dc.language.isoeng
dc.subjectKardiyoloji
dc.subjectDahili Tıp Bilimleri
dc.subjectSağlık Bilimleri
dc.subjectTıp
dc.subjectKlinik Tıp (MED)
dc.subjectKlinik Tıp
dc.subjectCARDIAC ve CARDIOVASCULAR SİSTEMLER
dc.titleThe relationship between endothelial nitric oxide synthase 4a/4b gene polymorphism and premature coronary artery disease
dc.typeMakale
dc.relation.journalACTA CARDIOLOGICA
dc.contributor.departmentDr. Siyami Ersek Cardiac & Vascular Surgery Training & Research Hospital , ,
dc.identifier.volume68
dc.identifier.issue5
dc.identifier.startpage464
dc.identifier.endpage468
dc.contributor.firstauthorID211215


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