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IL-1 beta is induced in reactive astrocytes in the somatosensory cortex of rats with genetic absence epilepsy at the onset of spike-and-wave discharges, and contributes to their occurrence

dc.contributor.authorAker, Rezzan Gulhan
dc.contributor.authorCarcak, Nihan
dc.contributor.authorVANZULLI, Ilaria
dc.contributor.authorVEZZANI, Annamaria
dc.contributor.authorOnat, Filiz Yilmaz
dc.contributor.authorAkin, Demet
dc.contributor.authorRAVIZZA, Teresa
dc.contributor.authorMAROSO, Mattia
dc.contributor.authorEryigit, Tugba
dc.date.accessioned2021-03-04T18:05:59Z
dc.date.available2021-03-04T18:05:59Z
dc.date.issued2011
dc.identifier.citationAkin D., RAVIZZA T., MAROSO M., Carcak N., Eryigit T., VANZULLI I., Aker R. G. , VEZZANI A., Onat F. Y. , "IL-1 beta is induced in reactive astrocytes in the somatosensory cortex of rats with genetic absence epilepsy at the onset of spike-and-wave discharges, and contributes to their occurrence", NEUROBIOLOGY OF DISEASE, cilt.44, ss.259-269, 2011
dc.identifier.issn0969-9961
dc.identifier.otherav_88ee8960-a674-4e58-8c92-e667db874166
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/92892
dc.identifier.urihttps://doi.org/10.1016/j.nbd.2011.05.015
dc.description.abstractInterleukin (IL)-1 beta plays a crucial role in the mechanisms of limbic seizures in rodent models of temporal lobe epilepsy. We addressed whether activation of the IL-1 beta signaling occurs in rats with genetic absence epilepsy (GAERS) during the development of spike-and-wave discharges (SWDs). Moreover, we studied whether inhibition of IL-1 beta biosynthesis in GAERS could affect SWD activity.
dc.language.isoeng
dc.subjectYaşam Bilimleri (LIFE)
dc.subjectTemel Bilimler
dc.subjectNEUROSCIENCES
dc.subjectSinirbilim ve Davranış
dc.subjectYaşam Bilimleri
dc.titleIL-1 beta is induced in reactive astrocytes in the somatosensory cortex of rats with genetic absence epilepsy at the onset of spike-and-wave discharges, and contributes to their occurrence
dc.typeMakale
dc.relation.journalNEUROBIOLOGY OF DISEASE
dc.contributor.departmentİstanbul Bilim Üniversitesi , ,
dc.identifier.volume44
dc.identifier.issue3
dc.identifier.startpage259
dc.identifier.endpage269
dc.contributor.firstauthorID82328


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