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LNCaP progression model of human prostate cancer: Androgen-independence and osseous metastasis

dc.contributor.authorChang, SM
dc.contributor.authorOzen, Mustafa
dc.contributor.authorDegeorges, A
dc.contributor.authorPathak, S
dc.contributor.authorChung, LWK
dc.contributor.authorThalmann, GN
dc.contributor.authorSikes, RA
dc.contributor.authorWu, TT
dc.date.accessioned2021-03-04T13:40:37Z
dc.date.available2021-03-04T13:40:37Z
dc.date.issued2000
dc.identifier.citationThalmann G., Sikes R., Wu T., Degeorges A., Chang S., Ozen M., Pathak S., Chung L., "LNCaP progression model of human prostate cancer: Androgen-independence and osseous metastasis", PROSTATE, cilt.44, sa.2, ss.91-103, 2000
dc.identifier.issn0270-4137
dc.identifier.othervv_1032021
dc.identifier.otherav_7dadbc89-2fba-4a8c-a1c1-b5a25343e084
dc.identifier.urihttp://hdl.handle.net/20.500.12627/85852
dc.description.abstractBACKGROUND. Clinically, the lethal phenotypes of human prostate cancer are characterized by their progression to androgen-independence and their propensity to form osseous metastases. We reported previously on the establishment of androgen-independent (AI) human prostate cancer cell lines derived from androgen-dependent (AD) LNCaP cells, with androgen independence defined as the capability of prostate cancer cells to grow in castrated hosts. One of the sublines, C4-2, was found to be AI, highly tumorigenic, and metastatic, having a proclivity for metastasis to the bone.
dc.language.isoeng
dc.subjectİç Hastalıkları
dc.subjectEndokrinoloji ve Metabolizma Hastalıkları
dc.subjectNefroloji
dc.subjectSağlık Bilimleri
dc.subjectDahili Tıp Bilimleri
dc.subjectTıp
dc.subjectÜROLOJİ VE NEFROLOJİ
dc.subjectKlinik Tıp (MED)
dc.subjectKlinik Tıp
dc.subjectENDOKRİNOLOJİ VE METABOLİZMA
dc.titleLNCaP progression model of human prostate cancer: Androgen-independence and osseous metastasis
dc.typeMakale
dc.relation.journalPROSTATE
dc.contributor.department, ,
dc.identifier.volume44
dc.identifier.issue2
dc.identifier.startpage91
dc.identifier.endpage103
dc.contributor.firstauthorID58445


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