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Mutation analysis of PRSS1, SPINK1 and CFTR gene in patients with alcoholic and idiopathic chronic pancreatitis: A single center study

dc.contributor.authorTugcu, Murat
dc.contributor.authorSisman, Gurhan
dc.contributor.authorŞENTÜRK, HAKAN
dc.contributor.authorAyla, Kerimova
dc.contributor.authorSebati, Ozdemir
dc.date.accessioned2021-03-04T13:11:51Z
dc.date.available2021-03-04T13:11:51Z
dc.date.issued2015
dc.identifier.citationSisman G., Tugcu M., Ayla K., Sebati O., ŞENTÜRK H., "Mutation analysis of PRSS1, SPINK1 and CFTR gene in patients with alcoholic and idiopathic chronic pancreatitis: A single center study", TURKISH JOURNAL OF GASTROENTEROLOGY, cilt.26, sa.2, ss.176-180, 2015
dc.identifier.issn1300-4948
dc.identifier.otherav_7b1b6865-6d01-44c4-bdf0-8f62916a9106
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/84310
dc.identifier.urihttps://doi.org/10.5152/tjg.2015.4287
dc.description.abstractBackground/Aims: A relation between some genetic mutations and chronic pancreatitis (CP) has been reported. However, the relation of genetic mutation to alcoholic CP (ACP) and idiopathic CP (ICP) still remains controversial. In this study, we investigated the prevalence of protease serine 1 (PRSS1), serine protease inhibitor, Kazal type 1 (SPINK1) SPINK1 and cystic fibrosis transmembrane conductance regulator (CFTR) mutations in ACP and ICP patients in Turkey.
dc.language.isoeng
dc.subjectSağlık Bilimleri
dc.subjectGastroenteroloji-(Hepatoloji)
dc.subjectDahili Tıp Bilimleri
dc.subjectİç Hastalıkları
dc.subjectTıp
dc.subjectKlinik Tıp (MED)
dc.subjectKlinik Tıp
dc.subjectGASTROENTEROLOJİ VE HEPATOLOJİ
dc.titleMutation analysis of PRSS1, SPINK1 and CFTR gene in patients with alcoholic and idiopathic chronic pancreatitis: A single center study
dc.typeMakale
dc.relation.journalTURKISH JOURNAL OF GASTROENTEROLOGY
dc.contributor.departmentİstanbul Üniversitesi , ,
dc.identifier.volume26
dc.identifier.issue2
dc.identifier.startpage176
dc.identifier.endpage180
dc.contributor.firstauthorID221302


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