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dc.contributor.authorSeven, A.
dc.contributor.authorBurcak, G.
dc.contributor.authorUzun, Hafize
dc.contributor.authorCivelek, S.
dc.contributor.authorAndican, G.
dc.contributor.authorAltunoglu, E.
dc.contributor.authorErdenen, F.
dc.contributor.authorŞahin, Ahmet Duran
dc.contributor.authorKutnu, M.
dc.date.accessioned2021-03-04T07:58:20Z
dc.date.available2021-03-04T07:58:20Z
dc.date.issued2015
dc.identifier.citationCivelek S., Kutnu M., Uzun H., Erdenen F., Altunoglu E., Andican G., Seven A., Şahin A. D. , Burcak G., "Soluble Lectin-Like Oxidized LDL Receptor 1 as a Possible Mediator of Endothelial Dysfunction in Patients With Metabolic Syndrome", JOURNAL OF CLINICAL LABORATORY ANALYSIS, cilt.29, sa.3, ss.184-190, 2015
dc.identifier.issn0887-8013
dc.identifier.otherav_60d8624f-f91b-48af-aa07-46f52aa621a3
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/67564
dc.identifier.urihttps://doi.org/10.1002/jcla.21748
dc.description.abstractBackgroundMetabolic syndrome (MetS) defines a well-known cluster of metabolic disturbances associated with an increased risk of cardiovascular disease and diabetes. The aim of this study was to examine the distribution of soluble lectin-like oxidized low-density lipoprotein (LDL) receptor-1 (sLOX-1) levels in patients with MetS, possible association of sLOX-1 with oxidized LDL (oxLDL), endothelial nitric oxide synthase (eNOS), nitric oxide (NOx), endothelin-1 (ET-1), paraoxonase 1 (PON1), and arylesterase (ARE) activities, and these parameters compared with healthy controls.
dc.language.isoeng
dc.subjectKlinik Tıp
dc.subjectKlinik Tıp (MED)
dc.subjectTıp
dc.subjectSağlık Bilimleri
dc.subjectTIBBİ LABORATUVAR TEKNOLOJİSİ
dc.titleSoluble Lectin-Like Oxidized LDL Receptor 1 as a Possible Mediator of Endothelial Dysfunction in Patients With Metabolic Syndrome
dc.typeMakale
dc.relation.journalJOURNAL OF CLINICAL LABORATORY ANALYSIS
dc.contributor.departmentIstanbul Training & Research Hospital , ,
dc.identifier.volume29
dc.identifier.issue3
dc.identifier.startpage184
dc.identifier.endpage190
dc.contributor.firstauthorID19335


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