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Simvastatin induces apoptosis in human breast cancer cells: p53 and estrogen receptor independent pathway requiring signalling through JNK

dc.contributor.authorAltiok, Nedret
dc.contributor.authorKoyuturk, Meral
dc.contributor.authorErsoz, Melike
dc.date.accessioned2021-03-03T21:01:51Z
dc.date.available2021-03-03T21:01:51Z
dc.date.issued2007
dc.identifier.citationKoyuturk M., Ersoz M., Altiok N., "Simvastatin induces apoptosis in human breast cancer cells: p53 and estrogen receptor independent pathway requiring signalling through JNK", CANCER LETTERS, cilt.250, sa.2, ss.220-228, 2007
dc.identifier.issn0304-3835
dc.identifier.othervv_1032021
dc.identifier.otherav_5d110b7f-c3bf-4eff-a16d-712848a684ae
dc.identifier.urihttp://hdl.handle.net/20.500.12627/65191
dc.identifier.urihttps://doi.org/10.1016/j.canlet.2006.10.009
dc.description.abstractThe effect of simvastatin, a widely used statin for the treatment of hypercholesterolemia, was investigated in the estrogen receptor (ER)-positive MCF-7, and the ER-negative MDA-MB 231 human breast cancer cell lines. Simvastatin induced cell cycle arrest and apoptosis in both cells. These effects of simvastatin were not altered by 17-beta-estradiol treatment.
dc.language.isoeng
dc.subjectDahili Tıp Bilimleri
dc.subjectSağlık Bilimleri
dc.subjectTıp
dc.subjectİç Hastalıkları
dc.subjectOnkoloji
dc.subjectKlinik Tıp (MED)
dc.subjectKlinik Tıp
dc.subjectONKOLOJİ
dc.titleSimvastatin induces apoptosis in human breast cancer cells: p53 and estrogen receptor independent pathway requiring signalling through JNK
dc.typeMakale
dc.relation.journalCANCER LETTERS
dc.contributor.department, ,
dc.identifier.volume250
dc.identifier.issue2
dc.identifier.startpage220
dc.identifier.endpage228
dc.contributor.firstauthorID182834


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