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dc.contributor.authorGul, A
dc.contributor.authorInanc, Murat
dc.contributor.authorMysercough, A
dc.contributor.authorOrdi-Ros, J
dc.contributor.authorOllier, W
dc.contributor.authorAlansari, A
dc.contributor.authorHajeer, A
dc.contributor.authorTeh, LS
dc.contributor.authorBayat, A
dc.date.accessioned2021-03-03T20:48:26Z
dc.date.available2021-03-03T20:48:26Z
dc.date.issued2002
dc.identifier.citationAlansari A., Hajeer A., Teh L., Bayat A., Mysercough A., Gul A., Inanc M., Ordi-Ros J., Ollier W., "Transforming growth factor-beta 2 polymorphism and systemic lupus erythematosus", JOURNAL OF RHEUMATOLOGY, cilt.29, sa.6, ss.1189-1191, 2002
dc.identifier.issn0315-162X
dc.identifier.othervv_1032021
dc.identifier.otherav_5bec4be5-de62-437a-a519-7c5e2d0b0a35
dc.identifier.urihttp://hdl.handle.net/20.500.12627/64472
dc.description.abstractObjective. To determine whether transforming growth factor-beta2 (TGF-beta2) gene polymorphism is associated with systemic lupus erythematosus (SLE) susceptibility. TGF-beta is a multifunctional family of cytokines important in tissue repair, inflammation and immunoregulation. SLE is thought to be a T cell dependent autoimmune disorder with T cell dysfunction. Due to its known suppressive effects on interleukin 2 dependent T cell growth, TGF-beta2 is considered to be a candidate SLE susceptibility gene. Furthermore, SLE has been linked with a region to which the TGF-B2 gene has been mapped.
dc.language.isoeng
dc.subjectİmmünoloji ve Romatoloji
dc.subjectSağlık Bilimleri
dc.subjectİç Hastalıkları
dc.subjectDahili Tıp Bilimleri
dc.subjectTıp
dc.subjectKlinik Tıp (MED)
dc.subjectKlinik Tıp
dc.subjectROMATOLOJİ
dc.titleTransforming growth factor-beta 2 polymorphism and systemic lupus erythematosus
dc.typeMakale
dc.relation.journalJOURNAL OF RHEUMATOLOGY
dc.contributor.department, ,
dc.identifier.volume29
dc.identifier.issue6
dc.identifier.startpage1189
dc.identifier.endpage1191
dc.contributor.firstauthorID165236


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