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Prokineticin receptor-1 induces neovascularization and epicardial-derived progenitor cell differentiation

dc.contributor.authorKurose, Hitoshi
dc.contributor.authorNebigil, Canan G.
dc.contributor.authorTakir, Selcuk
dc.contributor.authorMessaddeq, Nadia
dc.contributor.authorDierich, Andree
dc.contributor.authorUrayama, Kyoji
dc.contributor.authorGuilini, Celia
dc.contributor.authorTurkeri, Gulen
dc.date.accessioned2021-03-03T19:22:47Z
dc.date.available2021-03-03T19:22:47Z
dc.date.issued2008
dc.identifier.citationUrayama K., Guilini C., Turkeri G., Takir S., Kurose H., Messaddeq N., Dierich A., Nebigil C. G. , "Prokineticin receptor-1 induces neovascularization and epicardial-derived progenitor cell differentiation", ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, cilt.28, sa.5, ss.841-849, 2008
dc.identifier.issn1079-5642
dc.identifier.otherav_5433a10d-e140-444c-b878-a43dbf1dcef1
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/59653
dc.identifier.urihttps://doi.org/10.1161/atvbaha.108.162404
dc.description.abstractObjective-Identification of novel factors that contribute to myocardial repair and collateral vessel growth hold promise for treatment of heart diseases. We have shown that transient prokineticin receptor-1 (PKR1) gene transfer protects the heart against myocardial infarction in a mouse model. Here, we investigated the role of excessive PKR1 signaling in heart.
dc.language.isoeng
dc.subjectHematoloji
dc.subjectTıp
dc.subjectİç Hastalıkları
dc.subjectSağlık Bilimleri
dc.subjectDahili Tıp Bilimleri
dc.subjectPERİFERAL VASKÜLER HASTALIĞI
dc.subjectKlinik Tıp (MED)
dc.subjectKlinik Tıp
dc.subjectHEMATOLOJİ
dc.titleProkineticin receptor-1 induces neovascularization and epicardial-derived progenitor cell differentiation
dc.typeMakale
dc.relation.journalARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY
dc.contributor.departmentCentre National de la Recherche Scientifique (CNRS) , ,
dc.identifier.volume28
dc.identifier.issue5
dc.identifier.startpage841
dc.identifier.endpage849
dc.contributor.firstauthorID701691


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