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dc.contributor.authorYeni, Naz
dc.contributor.authorCoskun, Tulin
dc.contributor.authorOzkara, Cigdem
dc.contributor.authorKiziltan, Meral
dc.contributor.authorDelil, Sakir
dc.contributor.authorGunduz, Aysegul
dc.date.accessioned2021-03-03T15:33:19Z
dc.date.available2021-03-03T15:33:19Z
dc.identifier.citationCoskun T., Kiziltan M., Gunduz A., Delil S., Yeni N., Ozkara C., "Blink reflex in progressive myoclonic epilepsies", SEIZURE-EUROPEAN JOURNAL OF EPILEPSY, cilt.29, ss.169-173, 2015
dc.identifier.issn1059-1311
dc.identifier.otherav_3f91e2b4-e062-4dfd-9b7d-6b89e72c2b0a
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/46532
dc.identifier.urihttps://doi.org/10.1016/j.seizure.2015.05.006
dc.description.abstractPurpose: Progressive myoclonic epilepsies (PME) include a heterogeneous group of disorders. The brainstem is involved in these disorders, as demonstrated by neuroimaging and autopsy studies. The blink reflex (BR) is characteristically elicited after supraorbital electrical stimulation. The BR has two components, an ipsilateral R1 and bilateral R2 (R2 and R2c). The central generator of the BR is the brainstem. In this study, we aimed to investigate the functional status of the brainstem using the BR in PME cases with different etiological factors.
dc.language.isoeng
dc.subjectDahili Tıp Bilimleri
dc.subjectKLİNİK NEUROLOJİ
dc.subjectKlinik Tıp
dc.subjectKlinik Tıp (MED)
dc.subjectNEUROSCIENCES
dc.subjectSinirbilim ve Davranış
dc.subjectYaşam Bilimleri (LIFE)
dc.subjectTıp
dc.subjectSağlık Bilimleri
dc.subjectNöroloji
dc.subjectYaşam Bilimleri
dc.subjectTemel Bilimler
dc.titleBlink reflex in progressive myoclonic epilepsies
dc.typeMakale
dc.relation.journalSEIZURE-EUROPEAN JOURNAL OF EPILEPSY
dc.contributor.departmentİstanbul Üniversitesi , ,
dc.identifier.volume29
dc.identifier.startpage169
dc.identifier.endpage173
dc.contributor.firstauthorID29473


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