| dc.contributor.author | Onat, Altan | |
| dc.contributor.author | Can, Günay | |
| dc.date.accessioned | 2021-03-03T13:58:55Z | |
| dc.date.available | 2021-03-03T13:58:55Z | |
| dc.date.issued | 2014 | |
| dc.identifier.citation | Onat A., Can G., "Enhanced Proinflammatory State and Autoimmune Activation: a Breakthrough to Understanding Chronic Diseases", CURRENT PHARMACEUTICAL DESIGN, cilt.20, sa.4, ss.575-584, 2014 | |
| dc.identifier.issn | 1381-6128 | |
| dc.identifier.other | vv_1032021 | |
| dc.identifier.other | av_371e2c3b-3ded-480b-9981-1a6d0a324d81 | |
| dc.identifier.uri | http://hdl.handle.net/20.500.12627/41190 | |
| dc.identifier.uri | https://doi.org/10.2174/138161282004140213145551 | |
| dc.description.abstract | Insight is provided herein into the novel mechanisms of cardiometabolic risk. Previous reports, including the epidemiological work of the Turkish Adult Risk Factor study, indicated that proinflammatory state and oxidative stress are crucial for evaluating cardiometabolic risk. Autoimmune pathways in the course of oxidative stress are major determinants of cardiorenal and metabolic risk. The latter encompasses metabolic syndrome, type 2 diabetes, coronary heart disease, and chronic kidney disease (CKD). | |
| dc.language.iso | eng | |
| dc.subject | Temel Bilimler | |
| dc.subject | Yaşam Bilimleri | |
| dc.subject | Sağlık Bilimleri | |
| dc.subject | Yaşam Bilimleri (LIFE) | |
| dc.subject | Farmakoloji ve Toksikoloji | |
| dc.subject | FARMAKOLOJİ VE ECZACILIK | |
| dc.subject | Temel Eczacılık Bilimleri | |
| dc.subject | Eczacılık | |
| dc.title | Enhanced Proinflammatory State and Autoimmune Activation: a Breakthrough to Understanding Chronic Diseases | |
| dc.type | Makale | |
| dc.relation.journal | CURRENT PHARMACEUTICAL DESIGN | |
| dc.contributor.department | İstanbul Üniversitesi , , | |
| dc.identifier.volume | 20 | |
| dc.identifier.issue | 4 | |
| dc.identifier.startpage | 575 | |
| dc.identifier.endpage | 584 | |
| dc.contributor.firstauthorID | 90911 | |
