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Myofibrillar degeneration with diphtheria toxin

dc.contributor.authorEdis, Bilge
dc.contributor.authorNurten, Rustem
dc.contributor.authorBektas, Muhammet
dc.date.accessioned2021-03-02T17:27:21Z
dc.date.available2021-03-02T17:27:21Z
dc.date.issued2020
dc.identifier.citationEdis B., Bektas M., Nurten R., "Myofibrillar degeneration with diphtheria toxin", TURKISH JOURNAL OF BIOCHEMISTRY-TURK BIYOKIMYA DERGISI, cilt.45, ss.351-357, 2020
dc.identifier.issn0250-4685
dc.identifier.otherav_e4e4cc1a-762b-4026-b2c0-0ab5b1c2ca26
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/3798
dc.identifier.urihttps://doi.org/10.1515/tjb-2019-0109
dc.description.abstractObjectives: Cardiac damage in patient with diphtheritic myocarditis is reported as the leading cause of mortality. Diphtheria toxin (DTx) is a well-known bacterial toxin inducing various cytotoxic effects. Mainly, catalytic fragment inhibits protein synthesis, induces cytotoxicity, and depolymerizes actin filaments. In this study, we aimed to demonstrate the extent of myofibrillar damage under DTx treatment to porcine cardiac tissue samples.
dc.language.isoeng
dc.subjectStructural Biology
dc.subjectBİYOKİMYA VE MOLEKÜLER BİYOLOJİ
dc.subjectMoleküler Biyoloji ve Genetik
dc.subjectLife Sciences
dc.subjectTemel Bilimler
dc.subjectBiochemistry, Genetics and Molecular Biology (miscellaneous)
dc.subjectClinical Biochemistry
dc.subjectCancer Research
dc.subjectMolecular Biology
dc.subjectDrug Discovery
dc.subjectAging
dc.subjectGeneral Biochemistry, Genetics and Molecular Biology
dc.subjectBiochemistry
dc.subjectYaşam Bilimleri (LIFE)
dc.subjectYaşam Bilimleri
dc.subjectMoleküler Biyoloji ve Genetik
dc.subjectSitogenetik
dc.titleMyofibrillar degeneration with diphtheria toxin
dc.typeMakale
dc.relation.journalTURKISH JOURNAL OF BIOCHEMISTRY-TURK BIYOKIMYA DERGISI
dc.contributor.departmentİstanbul Teknik Üniversitesi , ,
dc.identifier.volume45
dc.identifier.issue4
dc.identifier.startpage351
dc.identifier.endpage357
dc.contributor.firstauthorID2358087


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