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dc.contributor.authorKORUYUCU, Mine
dc.contributor.authorLEE, M
dc.contributor.authorCHOI, M
dc.contributor.authorKim, JW
dc.contributor.authorHU, JC
dc.contributor.authorSIMMER, JP
dc.contributor.authorWang, SK
dc.contributor.authorHu, Y
dc.contributor.authorYANG, J
dc.contributor.authorSMITH, CE
dc.contributor.authorRichardson, AS
dc.contributor.authorYAMAKOSHI, Y
dc.contributor.authorLEE, YL
dc.contributor.authorSEYMEN, F
dc.contributor.authorGENCAY, K
dc.date.accessioned2021-03-03T09:52:41Z
dc.date.available2021-03-03T09:52:41Z
dc.date.issued2015
dc.identifier.citationWang S., Hu Y., YANG J., SMITH C., Richardson A., YAMAKOSHI Y., LEE Y., SEYMEN F., KORUYUCU M., GENCAY K., et al., "Fam83h null mice support a neomorphic mechanism for human ADHCAI.", MOLECULAR GENETICS AND GENOMICS, cilt.21, sa.4, ss.46-67, 2015
dc.identifier.issn1617-4615
dc.identifier.othervv_1032021
dc.identifier.otherav_1f4f1f6d-624b-4b86-a890-3f3cb527788c
dc.identifier.urihttp://hdl.handle.net/20.500.12627/26181
dc.language.isoeng
dc.titleFam83h null mice support a neomorphic mechanism for human ADHCAI.
dc.typeMakale
dc.relation.journalMOLECULAR GENETICS AND GENOMICS
dc.contributor.department, ,
dc.identifier.volume21
dc.identifier.issue4
dc.identifier.startpage46
dc.identifier.endpage67
dc.contributor.firstauthorID607646


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