dc.contributor.author | EŞKAZAN, AHMET EMRE | |
dc.contributor.author | EMRENCE, Zeliha | |
dc.contributor.author | ELVERDİ, TUĞRUL | |
dc.contributor.author | AR, MUHLİS CEM | |
dc.contributor.author | Salman Yaylaz, Burcu | |
dc.contributor.author | Ünüvar, Ayşegül | |
dc.contributor.author | Sarlman, Melda | |
dc.contributor.author | Gezer, Sümbül | |
dc.contributor.author | Karaman, Serap | |
dc.contributor.author | PAÇAL, Ferda | |
dc.contributor.author | Slrma-Ekmekci, Sema | |
dc.contributor.author | Abacl, Neslihan | |
dc.contributor.author | Karakaş, Zeynep | |
dc.contributor.author | Salihoǧlu, Ayşe | |
dc.date.accessioned | 2023-05-29T13:48:15Z | |
dc.date.available | 2023-05-29T13:48:15Z | |
dc.date.issued | 2023 | |
dc.identifier.citation | Gezer S., EMRENCE Z., ELVERDİ T., AR M. C., Salman Yaylaz B., PAÇAL F., Ünüvar A., Sarlman M., EŞKAZAN A. E., Karaman S., et al., "Elevación de SPINK2 en leucemia mieloide aguda", Advances in Laboratory Medicine, cilt.4, sa.1, ss.98-104, 2023 | |
dc.identifier.issn | 2628-491X | |
dc.identifier.other | av_3dc6904b-c38c-4951-b1b3-90ac6bdb70a4 | |
dc.identifier.other | vv_1032021 | |
dc.identifier.uri | http://hdl.handle.net/20.500.12627/189116 | |
dc.identifier.uri | https://doi.org/10.1515/almed-2023-0011 | |
dc.identifier.uri | https://avesis.istanbul.edu.tr/api/publication/3dc6904b-c38c-4951-b1b3-90ac6bdb70a4/file | |
dc.description.abstract | La leucemia mieloide aguda (AML), una patolog a maligna progresiva, es un trastorno de las c lulas madre hematopoy ticas caracterizada por un incremento en el n mero de c lulas mieloides de la m dula sea y el bloqueo de su maduraci n. Su incidencia aumenta con la edad [1, 2]. La AML es heterog nea en t rminos gen ticos, biol gicos y cl nicos. Algunas de las alteraciones gen ticas observadas en la AML consisten en alteraciones cromos micas, tales como la p rdida de parte o la totalidad de chr 5 y ch 7, t(15;17)(q22;q12), t(8;21)(q22;q22.1), inv(16)(p13.1q22)/ t(16;16)(p13.1;q22), inv(3)(q21.3q26.2)/t(3;3)(q21.3;q26.2), t(6;9)- (p23;q34.1), t(9;11)(p21.3;q23.3) y t(1;22)(p13.3;q13.3) [3]. Aparte de dichas alteraciones cromos micas, las mutaciones m s comunes se producen en los genes FLT3, NPM1, DNMT3A, KIT, CEBPA, RUNX1, IDH1/2 TET2 ve MLL, ASLX1, BCOR, BCORL1, TP53, GATA2, DDX41, KMT2A, SRP72 y STAG2 [4, 5] | |
dc.language.iso | eng | |
dc.subject | Tıp (çeşitli) | |
dc.subject | TIP, GENEL & DAHİLİ | |
dc.subject | EĞİTİM VE EĞİTİM ARAŞTIRMASI | |
dc.subject | Sosyal ve Beşeri Bilimler | |
dc.subject | Klinik Tıp (MED) | |
dc.subject | Sosyal Bilimler (SOC) | |
dc.subject | Klinik Tıp | |
dc.subject | Sosyal Bilimler Genel | |
dc.subject | Tıp | |
dc.subject | Sosyoloji | |
dc.subject | Eğitim | |
dc.subject | Sağlık Bilimleri | |
dc.subject | Temel Tıp Bilimleri | |
dc.subject | TIBBİ LABORATUVAR TEKNOLOJİSİ | |
dc.subject | Sosyal Bilimler ve Beşeri Bilimler | |
dc.subject | Tıbbi Laboratuvar Teknolojisi | |
dc.title | Elevación de SPINK2 en leucemia mieloide aguda | |
dc.type | Makale | |
dc.relation.journal | Advances in Laboratory Medicine | |
dc.contributor.department | İstanbul Üniversitesi , , | |
dc.identifier.volume | 4 | |
dc.identifier.issue | 1 | |
dc.identifier.startpage | 98 | |
dc.identifier.endpage | 104 | |
dc.contributor.firstauthorID | 4255159 | |