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Glutathione S-Transferase M1 and T1 Gene Deletions and Susceptibility to Acute Lymphoblastic Leukemia (ALL) in Adults

dc.contributor.authorZehra, Sitwat
dc.contributor.authorAzhar, Abid
dc.contributor.authorIsmail, Muhammad
dc.contributor.authorZehra, Alveena
dc.date.accessioned2022-02-18T11:25:39Z
dc.date.available2022-02-18T11:25:39Z
dc.date.issued2018
dc.identifier.citationZehra A., Zehra S., Ismail M., Azhar A., "Glutathione S-Transferase M1 and T1 Gene Deletions and Susceptibility to Acute Lymphoblastic Leukemia (ALL) in Adults", PAKISTAN JOURNAL OF MEDICAL SCIENCES, cilt.34, sa.3, ss.666-670, 2018
dc.identifier.issn1682-024X
dc.identifier.othervv_1032021
dc.identifier.otherav_f732e2a9-8926-43a0-afcc-40a016664fd2
dc.identifier.urihttp://hdl.handle.net/20.500.12627/181177
dc.identifier.urihttps://doi.org/10.12669/pjms.343.14911
dc.description.abstractObjective: Biotransformation of xenobiotics is critical for their metabolism and removal from the body which is carried out by xenobiotic metabolizing enzymes. Individuals carrying variants of genes that encode these enzymes have an altered ability to metabolize xenobiotics which may lead to an increased risk of acute lymphoblastic leukemia. The current study aimed to investigate the impact of GSTM1 and GSTT1 gene deletions in causing predisposition to adult ALL.
dc.language.isoeng
dc.subjectInternal Medicine
dc.subjectTemel Tıp Bilimleri
dc.subjectFamily Practice
dc.subjectFundamentals and Skills
dc.subjectGeneral Health Professions
dc.subjectPathophysiology
dc.subjectAssessment and Diagnosis
dc.subjectMedicine (miscellaneous)
dc.subjectGeneral Medicine
dc.subjectHealth Sciences
dc.subjectTIP, GENEL & İÇECEK
dc.subjectKlinik Tıp
dc.subjectKlinik Tıp (MED)
dc.subjectTıp
dc.subjectSağlık Bilimleri
dc.titleGlutathione S-Transferase M1 and T1 Gene Deletions and Susceptibility to Acute Lymphoblastic Leukemia (ALL) in Adults
dc.typeMakale
dc.relation.journalPAKISTAN JOURNAL OF MEDICAL SCIENCES
dc.contributor.departmentUniversity of Karachi , ,
dc.identifier.volume34
dc.identifier.issue3
dc.identifier.startpage666
dc.identifier.endpage670
dc.contributor.firstauthorID3386221


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