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DNA Damage in AML-12 Hepatocytes and 3T3-L1 Adipocytes Treated with Clopidogrel

dc.contributor.authorCevik, Mehtap
dc.contributor.authorCagatay, Penbe
dc.contributor.authorCaker, Selen
dc.contributor.authorBayar, Elif
dc.contributor.authorSÜSLEYİCİ, BELGİN
dc.date.accessioned2022-02-18T09:30:26Z
dc.date.available2022-02-18T09:30:26Z
dc.date.issued2021
dc.identifier.citationBayar E., Cevik M., Caker S., Cagatay P., SÜSLEYİCİ B., "DNA Damage in AML-12 Hepatocytes and 3T3-L1 Adipocytes Treated with Clopidogrel", CURRENT DRUG SAFETY, cilt.16, sa.3, ss.252-258, 2021
dc.identifier.otherav_426e1be0-5722-4feb-b00d-a6e13cb6d73b
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/177366
dc.identifier.urihttps://doi.org/10.2174/1574886315666210106141936
dc.description.abstractBackground: Clopidogrel has been commonly prescribed as a selective P2Y(12) receptor antagonist to reduce heart attack and stroke risk. Nearly 10% of absorbed clopidogrel is metabolized to active forms by cytochrome P450 (CYP) enzymes in the liver and 90% to inactive clopidogrel carboxylate by esterases.
dc.language.isoeng
dc.subjectEczacılık
dc.subjectTemel Eczacılık Bilimleri
dc.subjectYaşam Bilimleri
dc.subjectTemel Bilimler
dc.subjectPharmacology
dc.subjectGeneral Pharmacology, Toxicology and Pharmaceutics
dc.subjectPharmacology, Toxicology and Pharmaceutics (miscellaneous)
dc.subjectPharmacology (medical)
dc.subjectPharmacy
dc.subjectDrug Guides
dc.subjectLife Sciences
dc.subjectHealth Sciences
dc.subjectFARMAKOLOJİ VE ECZACILIK
dc.subjectYaşam Bilimleri (LIFE)
dc.subjectFarmakoloji ve Toksikoloji
dc.subjectSağlık Bilimleri
dc.titleDNA Damage in AML-12 Hepatocytes and 3T3-L1 Adipocytes Treated with Clopidogrel
dc.typeMakale
dc.relation.journalCURRENT DRUG SAFETY
dc.contributor.departmentMarmara Üniversitesi , ,
dc.identifier.volume16
dc.identifier.issue3
dc.identifier.startpage252
dc.identifier.endpage258
dc.contributor.firstauthorID2774071


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