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Impact of UCP2 -866G/A Variant on Smoking Risk

dc.contributor.authorSever, Ulgen
dc.contributor.authorPehlivan, Sacide
dc.contributor.authorNURSAL, AYŞE FEYDA
dc.contributor.authorUysal, Mehmet Atilla
dc.contributor.authorPEHLİVAN, MUSTAFA
dc.date.accessioned2021-12-10T09:41:52Z
dc.date.available2021-12-10T09:41:52Z
dc.date.issued2021
dc.identifier.citationNURSAL A. F. , Uysal M. A. , PEHLİVAN M., Sever U., Pehlivan S., "Impact of UCP2 -866G/A Variant on Smoking Risk", BEZMIALEM SCIENCE, cilt.9, sa.2, ss.185-189, 2021
dc.identifier.otherav_0cc305c0-095d-4c3a-b133-e1595038e7e0
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/168266
dc.identifier.urihttps://doi.org/10.14235/bas.galenos.2020.4159
dc.description.abstractObjective: Mitochondria are multifunctional and dynamic organelles found in cells. Nicotine is a natural alkaloid found in the tobacco plant and has been well studied as a component of cigarette smoke. It has also been reported to affect mitochondrial function both in vitro and in vivo. Uncoupling protein 2 (UCP2) reduces generation of ROS by mitochondria. Our purpose in this study was to investigate whether the -866G/A variant of the UCP2 gene is associated with smoking status.
dc.language.isoeng
dc.subjectInternal Medicine
dc.subjectTIP, GENEL & İÇECEK
dc.subjectKlinik Tıp
dc.subjectKlinik Tıp (MED)
dc.subjectTıp
dc.subjectSağlık Bilimleri
dc.subjectTemel Tıp Bilimleri
dc.subjectFamily Practice
dc.subjectFundamentals and Skills
dc.subjectGeneral Health Professions
dc.subjectPathophysiology
dc.subjectAssessment and Diagnosis
dc.subjectMedicine (miscellaneous)
dc.subjectGeneral Medicine
dc.subjectHealth Sciences
dc.titleImpact of UCP2 -866G/A Variant on Smoking Risk
dc.typeMakale
dc.relation.journalBEZMIALEM SCIENCE
dc.contributor.departmentHitit Üniversitesi , Tıp Fakültesi , Dahili Tıp Bilimleri Bölümü
dc.identifier.volume9
dc.identifier.issue2
dc.identifier.startpage185
dc.identifier.endpage189
dc.contributor.firstauthorID2633217


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