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Polymorphisms of endothelin 1 (G5665T and T-1370G) and endothelin receptor type A (C+70G and G-231A) in Graves' disease

dc.contributor.authorVural, Pervin
dc.contributor.authorAydın, Ali Fuat
dc.contributor.authorKARADAG, Berrin
dc.contributor.authorUysal, Mujdat
dc.contributor.authorDogru-Abbasoglu, Semra
dc.contributor.authorDeveli-Is, Seval
dc.contributor.authorOZDERYA, Aysenur
dc.date.accessioned2021-03-06T21:07:13Z
dc.date.available2021-03-06T21:07:13Z
dc.date.issued2014
dc.identifier.citationAydın A. F. , Develi-Is S., Dogru-Abbasoglu S., Vural P., OZDERYA A., KARADAG B., Uysal M., "Polymorphisms of endothelin 1 (G5665T and T-1370G) and endothelin receptor type A (C+70G and G-231A) in Graves' disease", INTERNATIONAL IMMUNOPHARMACOLOGY, cilt.18, ss.198-202, 2014
dc.identifier.issn1567-5769
dc.identifier.otherav_fd44f033-4b5e-4719-8e1b-995332a40d18
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/165695
dc.identifier.urihttps://doi.org/10.1016/j.intimp.2013.11.017
dc.description.abstractPurpose: Endothelin 1 (EDN1) is a strong angiogenic and mitogenic factor, playing a key role in hypervascularization, thyroid follicle cell hyperplasia, and lymphocyte infiltration in the thyroid gland of patients with Graves' disease (GD). EDN1 induces angiogenesis and mitogenesis via endothelin receptor type A (EDNRA). This study examined the possible association of EDN1 (G5665T and T-1370G) and EDNRA (C+ 70G and G-231A) single nucleotide polymorphisms (SNPs) with the occurrence of GD, and evaluates the relationship between genotypes and clinical/laboratory manifestations of GD.
dc.language.isoeng
dc.subjectTemel Bilimler
dc.subjectSağlık Bilimleri
dc.subjectYaşam Bilimleri
dc.subjectEczacılık
dc.subjectTemel Eczacılık Bilimleri
dc.subjectFarmakoloji ve Toksikoloji
dc.subjectFARMAKOLOJİ VE ECZACILIK
dc.subjectYaşam Bilimleri (LIFE)
dc.subjectİmmünoloji
dc.titlePolymorphisms of endothelin 1 (G5665T and T-1370G) and endothelin receptor type A (C+70G and G-231A) in Graves' disease
dc.typeMakale
dc.relation.journalINTERNATIONAL IMMUNOPHARMACOLOGY
dc.contributor.departmentİstanbul Üniversitesi , ,
dc.identifier.volume18
dc.identifier.issue1
dc.identifier.startpage198
dc.identifier.endpage202
dc.contributor.firstauthorID44549


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