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dc.contributor.authorCandan, G
dc.contributor.authorYigit, G
dc.contributor.authorHatemi, H
dc.contributor.authorAndican, G
dc.contributor.authorSimsek, G
dc.contributor.authorKarakoc, Y
dc.date.accessioned2021-03-06T20:06:32Z
dc.date.available2021-03-06T20:06:32Z
dc.date.issued1997
dc.identifier.citationSimsek G., Andican G., Karakoc Y., Yigit G., Hatemi H., Candan G., "Calcium, magnesium, and zinc status in experimental hypothyroidism", BIOLOGICAL TRACE ELEMENT RESEARCH, cilt.60, ss.205-213, 1997
dc.identifier.issn0163-4984
dc.identifier.otherav_f8e3e918-de73-4875-922e-89eaa01df6e9
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/163024
dc.identifier.urihttps://doi.org/10.1007/bf02784440
dc.description.abstractIn this study, experimental hypothyroidism was established and used to investigate possible alterations in the calcium, magnesium, and zinc homeostasis by assessing their concentration in plasma and erythrocytes. Hypothyroidism was induced by administration of methimazole an iodine blocker at a dose of 75 mg/100 g food for 3 wk.
dc.language.isoeng
dc.subjectSağlık Bilimleri
dc.subjectBİYOKİMYA VE MOLEKÜLER BİYOLOJİ
dc.subjectMoleküler Biyoloji ve Genetik
dc.subjectYaşam Bilimleri (LIFE)
dc.subjectENDOKRİNOLOJİ VE METABOLİZMA
dc.subjectKlinik Tıp
dc.subjectKlinik Tıp (MED)
dc.subjectTıp
dc.subjectDahili Tıp Bilimleri
dc.subjectİç Hastalıkları
dc.subjectEndokrinoloji ve Metabolizma Hastalıkları
dc.subjectYaşam Bilimleri
dc.subjectMoleküler Biyoloji ve Genetik
dc.subjectSitogenetik
dc.subjectTemel Bilimler
dc.titleCalcium, magnesium, and zinc status in experimental hypothyroidism
dc.typeMakale
dc.relation.journalBIOLOGICAL TRACE ELEMENT RESEARCH
dc.contributor.department, ,
dc.identifier.volume60
dc.identifier.issue3
dc.identifier.startpage205
dc.identifier.endpage213
dc.contributor.firstauthorID119894


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