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dc.contributor.authorUysal, Mujdat
dc.contributor.authorBekpinar, Seldağ
dc.contributor.authorGurdol, Figen
dc.contributor.authorUnlucerci, Yesim
dc.date.accessioned2021-03-06T09:45:15Z
dc.date.available2021-03-06T09:45:15Z
dc.date.issued2014
dc.identifier.citationBekpinar S., Unlucerci Y., Uysal M., Gurdol F., "Propargylglycine aggravates liver damage in LPS-treated rats: Possible relation of nitrosative stress with the inhibition of H2S formation", PHARMACOLOGICAL REPORTS, cilt.66, ss.897-901, 2014
dc.identifier.issn1734-1140
dc.identifier.othervv_1032021
dc.identifier.otherav_e70208b6-30d9-4cd9-b6af-ff7501102888
dc.identifier.urihttp://hdl.handle.net/20.500.12627/151945
dc.identifier.urihttps://doi.org/10.1016/j.pharep.2014.05.014
dc.description.abstractBackground: Hydrogen sulfide (H2S) is a naturally occurring gaseous transmitter, which may play important roles in normal physiology and disease. Here, we investigated the effect of endogenously formed H2S in the endotoxemic organ injury.
dc.language.isoeng
dc.subjectTemel Eczacılık Bilimleri
dc.subjectYaşam Bilimleri
dc.subjectTemel Bilimler
dc.subjectSağlık Bilimleri
dc.subjectEczacılık
dc.subjectYaşam Bilimleri (LIFE)
dc.subjectFarmakoloji ve Toksikoloji
dc.subjectFARMAKOLOJİ VE ECZACILIK
dc.titlePropargylglycine aggravates liver damage in LPS-treated rats: Possible relation of nitrosative stress with the inhibition of H2S formation
dc.typeMakale
dc.relation.journalPHARMACOLOGICAL REPORTS
dc.contributor.departmentİstanbul Üniversitesi , ,
dc.identifier.volume66
dc.identifier.issue5
dc.identifier.startpage897
dc.identifier.endpage901
dc.contributor.firstauthorID29801


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