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dc.contributor.authorOzbek, Uğur
dc.contributor.authorErsoy, Ismail Hakki
dc.contributor.authorCagatay, Penbe
dc.contributor.authorKadioglu, Pınar
dc.contributor.authorSayıtıoglu, Müge
dc.contributor.authorCiplak, Nejat
dc.contributor.authorErensoy, Nevin
dc.contributor.authorHatımaz, Ozden
dc.contributor.authorKısacık, Bunyamin
dc.contributor.authorOz, Buge
dc.contributor.authorSar, Mehmet
dc.contributor.authorGazioglu, Nurperi
dc.date.accessioned2021-03-02T22:20:58Z
dc.date.available2021-03-02T22:20:58Z
dc.identifier.citationGazioglu N., Erensoy N., Kadioglu P., Sayıtıoglu M., Ersoy I. H. , Hatımaz O., Kısacık B., Oz B., Sar M., Ozbek U., et al., "Altered cyclin D1 genotype distribution in human sporadic pituitary adenomas.", Medical science monitor : international medical journal of experimental and clinical research, cilt.13, 2007
dc.identifier.issn1234-1010
dc.identifier.othervv_1032021
dc.identifier.otherav_0c2a56c6-bb52-4c40-8934-f1811bba2b8d
dc.identifier.urihttp://hdl.handle.net/20.500.12627/13826
dc.description.abstractBackground: The cyclin D1 gene (CCND1) is a proto-oncogene playing a critical role in the transition through the G1 ot the S phase of the cell cyle and is overexpressed in many tumors. G870A plymorphism at the exon4/intron4 splicing region of the CCND1 gene may play a role in pituitary tumorigenesis and invasiveness. The objective of this study was to examine CCND1 polymorphism i patients with different types sporadic pituitary adenomas.
dc.language.isoeng
dc.subjectTıbbi Ekoloji ve Hidroklimatoloji
dc.subjectSağlık Bilimleri
dc.subjectDahili Tıp Bilimleri
dc.subjectTıp
dc.subjectKlinik Tıp (MED)
dc.subjectKlinik Tıp
dc.subjectTIP, ARAŞTIRMA VE DENEYSEL
dc.titleAltered cyclin D1 genotype distribution in human sporadic pituitary adenomas.
dc.typeMakale
dc.relation.journalMedical science monitor : international medical journal of experimental and clinical research
dc.contributor.departmentİstanbul Üniversitesi , ,
dc.identifier.volume13
dc.identifier.endpage63
dc.contributor.firstauthorID1040922


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