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dc.contributor.authorAYDIN OMAY, BANU
dc.contributor.authorOnat, Filiz
dc.contributor.authorRaci Yananli, Hasan
dc.contributor.authorAkman, Ozlem
dc.contributor.authorCarcak, Nihan
dc.contributor.authorYAVUZ, Melis
dc.date.accessioned2021-03-05T18:57:21Z
dc.date.available2021-03-05T18:57:21Z
dc.identifier.citationYAVUZ M., AYDIN OMAY B., Carcak N., Akman O., Raci Yananli H., Onat F., "Atipamezole, a specific alpha(2A) antagonist, suppresses spike-and-wave discharges and alters Ca2+/calmodulin-dependent protein kinase II in the thalamus of genetic absence epilepsy rats", EPILEPSIA, 2020
dc.identifier.issn0013-9580
dc.identifier.otherav_cce66147-2772-4d79-9ba4-d855ee27b73e
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/135638
dc.identifier.urihttps://doi.org/10.1111/epi.16728
dc.description.abstractObjective The role of alpha(2A) adrenergic receptors (alpha(2A)ARs) in absence epilepsy is not well characterized. Therefore, we investigated the outcomes of the specific antagonism of alpha(2A)ARs on the spike-and-wave discharges (SWDs) in genetic absence epilepsy rats from Strasbourg (GAERSs), together with its influence on the behavior and second messenger systems, which may point to the mechanisms to which a possible SWD modulation can be related.
dc.language.isoeng
dc.subjectTıp
dc.subjectDahili Tıp Bilimleri
dc.subjectNöroloji
dc.subjectSağlık Bilimleri
dc.subjectKLİNİK NEUROLOJİ
dc.subjectKlinik Tıp
dc.subjectKlinik Tıp (MED)
dc.titleAtipamezole, a specific alpha(2A) antagonist, suppresses spike-and-wave discharges and alters Ca2+/calmodulin-dependent protein kinase II in the thalamus of genetic absence epilepsy rats
dc.typeMakale
dc.relation.journalEPILEPSIA
dc.contributor.departmentAcıbadem Mehmet Ali Aydınlar Üniversitesi , Eczacılık Fakültesi , Eczacılık Meslek Birimleri Bölümü
dc.contributor.firstauthorID2359403


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