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Lack of association between the IL1A gene (-889) polymorphism and outcome after head injury

dc.contributor.authorBaykara, O
dc.contributor.authorUzan, M
dc.contributor.authorOzkara, C
dc.contributor.authorBuyra, N
dc.contributor.authorIs, M
dc.contributor.authorTanriverdi, T
dc.contributor.authorSanus, GZ
dc.date.accessioned2021-03-02T21:37:58Z
dc.date.available2021-03-02T21:37:58Z
dc.date.issued2006
dc.identifier.citationTanriverdi T., Uzan M., Sanus G., Baykara O., Is M., Ozkara C., Buyra N., "Lack of association between the IL1A gene (-889) polymorphism and outcome after head injury", SURGICAL NEUROLOGY, cilt.65, sa.1, ss.7-10, 2006
dc.identifier.issn0090-3019
dc.identifier.otherav_080ea5f3-c45d-4e16-846c-485b60b8a6a8
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/11231
dc.identifier.urihttps://doi.org/10.1016/j.surneu.2005.05.024
dc.description.abstractBackground: Interleukin (IL) I is a proinflammatory cytokine that has been identified as an important mediator of neurodegeneration induced by ischemia or traumatic brain injury. Accumulating evidence to date has suggested that the major cytokine contributing to neurodegeneration after head injury is IL-1 beta rather than IL-1 alpha; however, there is no sufficient data regarding IL-1 alpha in literature, and there may be an association between IL1A gene polymorphism and outcome after head injury.
dc.language.isoeng
dc.subjectDahili Tıp Bilimleri
dc.subjectNöroloji
dc.subjectCerrahi Tıp Bilimleri
dc.subjectKLİNİK NEUROLOJİ
dc.subjectKlinik Tıp
dc.subjectKlinik Tıp (MED)
dc.subjectCERRAHİ
dc.subjectTıp
dc.subjectSağlık Bilimleri
dc.titleLack of association between the IL1A gene (-889) polymorphism and outcome after head injury
dc.typeMakale
dc.relation.journalSURGICAL NEUROLOGY
dc.contributor.department, ,
dc.identifier.volume65
dc.identifier.issue1
dc.identifier.startpage7
dc.identifier.endpage10
dc.contributor.firstauthorID177439


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