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MAPK overexpression is associated with anthracycline resistance and increased risk for recurrence in patients with triple-negative breast cancer

dc.contributor.authorSaip, P.
dc.contributor.authorEralp, Y.
dc.contributor.authorGuney, N.
dc.contributor.authorTopuz, E.
dc.contributor.authorDerin, D.
dc.contributor.authorYavuz, Erdem
dc.contributor.authorIgci, A.
dc.contributor.authorMuslumanoglu, M.
dc.contributor.authorKucucuk, S.
dc.contributor.authorDincer, M.
dc.contributor.authorAydiner, A.
dc.contributor.authorOzluk, Y.
dc.date.accessioned2021-03-05T11:14:50Z
dc.date.available2021-03-05T11:14:50Z
dc.date.issued2008
dc.identifier.citationEralp Y., Derin D., Ozluk Y., Yavuz E., Guney N., Saip P., Muslumanoglu M., Igci A., Kucucuk S., Dincer M., et al., "MAPK overexpression is associated with anthracycline resistance and increased risk for recurrence in patients with triple-negative breast cancer", ANNALS OF ONCOLOGY, cilt.19, ss.669-674, 2008
dc.identifier.issn0923-7534
dc.identifier.othervv_1032021
dc.identifier.otherav_a73a3d46-e4eb-495c-88f4-ec07c765b2b6
dc.identifier.urihttp://hdl.handle.net/20.500.12627/111802
dc.identifier.urihttps://doi.org/10.1093/annonc/mdm522
dc.description.abstractBackground: Triple-negative breast cancer is estimated to account for 15%-20% of all patients with breast cancer and is considered as a prognostically unfavorable subset. The aim of this study is to evaluate the prognostic impact of various molecular factors in patients with triple-negative breast cancer.
dc.language.isoeng
dc.subjectDahili Tıp Bilimleri
dc.subjectOnkoloji
dc.subjectİç Hastalıkları
dc.subjectONKOLOJİ
dc.subjectKlinik Tıp
dc.subjectKlinik Tıp (MED)
dc.subjectTıp
dc.subjectSağlık Bilimleri
dc.titleMAPK overexpression is associated with anthracycline resistance and increased risk for recurrence in patients with triple-negative breast cancer
dc.typeMakale
dc.relation.journalANNALS OF ONCOLOGY
dc.contributor.departmentİstanbul Teknik Üniversitesi , Fen-Edebiyat , Kimya
dc.identifier.volume19
dc.identifier.issue4
dc.identifier.startpage669
dc.identifier.endpage674
dc.contributor.firstauthorID72350


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