Yazar "Eraksoy, Mefküre" için Avesis listeleme
-
A genome screen for linkage disequilibrium in Turkish multiple sclerosis
Eraksoy, Mefküre (2003)A genome screen for linkage disequilibrium in Turkish multiple sclerosisM. Eraksoya,*,1, A. Hensiekb,1, M. Kurtuncua,b, G. Akman-Demira, M. Kılıncc,M. Gedizlioglud, B. Petek-Balcıe, O¨ . Anlarf, C. Kutlug, G. Saruhan-Dir ... -
A whole genome screen for linkage in Turkish multiple sclerosis
Eraksoy, Mefküre (2003)A whole genome screen for linkage in Turkish multiple sclerosisM. Eraksoya,*,1, M. Kurtuncua,b,1, G. Akman-Demira, M. Kılıncc, M. Gedizlioglud, M. Mirzae,O¨. Anlarf, C. Kutlug, M. Demirkıranh, H.A. I˙drisoglua, A. Compstonb, ... -
Bilateral demyelinating tumefactive lesions in three children with hemiparesis
Yapici, Z; Eraksoy, Mefküre (2002)We present the results from the evaluations of three children ages of 2, 7, and 11 years with hemiparesis and multiple white-matter lesions on magnetic resonance images (MRIs). The initial symptoms were mainly acute/subacute ... -
HLA-DR and -DQ Associations with Multiple Sclerosis in Turkey
Eraksoy, Mefküre; Baykal, Betül; Saruhan Direskeneli, Güher (1997)ABSTRACT: The DRB, DQA, and DQB subregions ofthe major histocompatibility complex (MHC) were investigatedby polymerase chain reaction and sequence-specificoligonucleotide probe hybridization (PCR/SSO) in103 multiple sclerosis ... -
Non-pProgressive congenital ataxia with cerebellar hypoplasia in three families.
Yapıcı, Zuhal; Eraksoy, Mefküre (2005)AbstractAim: Non-progressive ataxias with cerebellar hypoplasia are a rarely seen heterogeneous group of hereditary cerebellar ataxias.Method: Three sib pairs from three different families with this entity have been reviewed, ... -
Proton spectroscopic findings in children with epilepsy owing to tuberous sclerosis complex.
Yapıcı, Zuhal; Eraksoy, Mefküre; Dinçer, Alp (2005)ABSTRACTTuberous sclerosis complex is an autosomal dominant disorder that often causes refractory seizures. The presence of multiplelesions makes it difficult to identify a single lesion responsible for the epilepsy. Our ...